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In nerves uncovered to 100 Hz when bathed in 10 mM glucose, the CAP failed suggesting that even within the presence of normoglycemic glucose additional energy substrate is required when the tissue power demand increases. Such knowledge are necessary as they show that glycogen has an necessary function under physiological circumstances. The fact that glycogen supported CAP conduction within the absence of exogenously utilized glucose in all probability has no worth in ascribing a task to glycogen, since aglycaemia is an artificial experimental process, and hypoglycemia is an iatrogenic consequence of insulin therapy that did not exist previous to 1922 (Frier et al., 2014), thus cannot be glycogen’s essential function. However supplying lactate to axons when tissue vitality demand increases implies a novel function for mind glycogen and suggests it acts as an vitality buffer slightly than an power reserve, whose function is not to single-handedly help conduction, however to act in live performance with ambient levels of normoglycemic glucose.

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